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Showing posts from February, 2010

CELL INJURY AND NECROSIS

INTRODUCTION AND OBJECTIVES: In this unit, we explore basic ideas about what "diseases" are, how they are produced, and how they are recognized. Details and further explanations are available in Chapter I of Robbins. Chapter I deals with cellular injury and cellular reactions to injury. "Injury" refers to the various agents and modalities that act on cells (such as, chemicals, toxins, organisms, intracellular accumulations, temperature changes, radiation, etc.). Cells can react by modifying themselves slightly and thus adapt to the injury (such as hypertrophy, hyperplasia, atrophy, and metaplasia, hydropic swelling, and fatty change). Such adaptations may be reversible. Cells can also react by becoming permanently altered and then living a new "lifestyle" (such as radiation changes in cells). They may also react to injury by being overwhelmed, and unable to continue life, and so they die. When cells die, they can do so in a pre- and proscribed...

MICROBIOLOGY LECTURES

TO DOWNLOAD A LECTURE CLICK ON X mark AGAINST THE TOPIC Lecture PPT Introduction to MMID X Intro I: virus structure and classification X Intro II: laboratory virology X Intro III: pathogenesis & genetics X DNA viruses I: papillomaviruses X DNA viruses II: adeno, parvo and polyomaviruses X (-) RNA viruses I: influenza virus X (-) RNA viruses II: measles, mumps, parainfluenza and respiratory syncytial viruses X (-) RNA viruses III: rhabdovirus (rabies) and reoviruses (rotavirus, diarrhea) X (+) RNA viruses I: ente...